In the presence of iron, lipid peroxides are transformed into toxic lipid free radicals (LO-), which cause the breakdown of polyunsaturated fatty acids in cell membrane lipids and cause cell death [2]. Iron death as a stress response has been observed in a variety of pathological conditions, including neurodegenerative diseases, ischemia-reperfusion injury, iron metabolism-related diseases, and tumors [3]In the presence of iron, lipid peroxides are transformed into toxic lipid free radicals (LO-), which cause the breakdown of polyunsaturated fatty acids in cell membrane lipids and cause cell death [2]. Iron death as a stress response has been observed in a variety of pathological conditions, including neurodegenerative diseases, ischemia-reperfusion injury, iron metabolism-related diseases, and tumors [3]In the presence of iron, lipid peroxides are transformed into toxic lipid free radicals (LO-), which cause the breakdown of polyunsaturated fatty acids in cell membrane lipids and cause cell death [2]. Iron death as a stress response has been observed in a variety of pathological conditions, including neurodegenerative diseases, ischemia-reperfusion injury, iron metabolism-related diseases, and tumors [3]